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Importantly, co-infection of H. Even single infections with H. Endolarasitarias study points out yahpo need to study both pathogens to understand the mechanism behind severe damage of the gastric mucosa, which could identified children with increased risk to present more serious lesions later in life. December 26, ; Accepted: March 26, ; Published: April 24, Copyright: This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.
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Competing interests: The authors have declared that no competing interests exist. Introduction Persistent infections often lead endoparaistarias chronic inflammation, a well documented cancer risk factor. Gastric cancer GC Enfermeades starts with an inflammatory process mainly associated with infection by Helicobacter pylori H. GC is the Enfermedaxes most common type of cancer and the second cause of death by cancer world-wide, affecting particularly Asian and Latin American countries . More recently, GC has also been associated with Epstein-Barr virus EBV but the role of the viral infection in early inflammatory gastric responses remains poorly studied.
Inflammation after infection in children is usually associated with a low level of polymorphonuclear PMN and mononuclear MN cells infiltrating the gastric mucosa . It has been suggested than the earlier the infection, the greater the risk to present GC later in life, conceivable because of a long lasting decades chronic inflammatory reaction to the infection . Only a fraction of H.
The outcome of H. Among the most important bacterial virulence factors is the pathogenicity island CagPAIwhich encodes a type IV secretion system T4SS that translocates the effector protein CagA into epithelial cells . CagA activates multiple signaling pathways triggering cellular phenotypes associated with oncogenic transformation . Moreover, transgenic mice expressing CagA develop adenocarcinomas of the digestive tract. Based on these data, CagA has been recognized as the first known bacterial oncoprotein . EBV infection has been consistently associated with several types of lymphoma, nasopharyngeal carcinoma NPC  and more recently to GC .
EBV infection also occurs early in childhood and usually persists in B cells, with most infected individuals carrying the virus asymptomatically in a latent stage in these cells. It is not clear when EBV infects the gastric mucosa and whether infection induces an inflammatory reaction, as observed with H. EBV reactivation from infected B cells has been proposed to facilitate infection of the epithelial basolateral face . In that scenario, the titer of anti-EBV antibodies against structural proteins has been proposed to correlate with the level of viral reactivation and as a prognostic marker in NPC .
To our knowledge, no studies have previously addressed whether EBV infection in children is associated with inflammation in the gastric mucosa or whether there exists a cooperative effect between EBV and H. In this study, we analyzed antibodies against EBV and H.
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Our results strongly suggest that single infection by either EBV or H. Even infection with H. These data argue for a previously unknown critical role of EBV infection in the induction of an inflammatory response in the gastric mucosa of children. Parents or guardians of the patients were informed on the nature of the study and those willing to participate signed a written informed consent prior to specimen collection. Overview Study population. It looks like Serena Williams is taking part in a new type of tennis match. Don't be enfermedades endoparasitarias yahoo dating kiss ass.
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